@article{McCracken_I-2021_118337,
title = {Lack of evidence of ACE2 expression and replicative infection by SARS-CoV-2 in human endothelial cells},
author = {McCracken, I. and Saginc, G. and He, L. and Huseynov, A. and Daniels, A. and Fletcher, S. and Peghaire, C. and Kalna, V. and Andaloussi-mäe, M. and Muhl, L. and Craig, N. and Griffiths, S. and Haas, J. and Tait-Burkard, C. and Lendahl, U. and Birdsey, G. and Betsholtz, C. and Noseda, M. and Baker, A. and Randi, A.},
month = {jan},
year = {2021},
abstract = {A striking feature of severe forms of coronavirus disease 2019 (COVID-19), the current pandemic caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), is severe endothelial injury with micro- and macrothrombotic disease in the lung and other organs, including the heart. This has led to speculation that viral infection may damage the endothelium through 2 mechanisms: indirectly, via neighborhood effects, circulating mediators, and immune mechanisms or directly by viral infection of endothelial cells (ECs).},
pages = {865-868},
volume = {143},
issue = {8},
journal = {Circulation},
publisher = {American Heart Association},
url = {https://doi.org/10.1161/CIRCULATIONAHA.120.052824},
}
